Atherosclerosis

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β-Cryptoxanthin and the risk for lifestyle-related disease: findings from recent nutritional epidemiologic studies.

​A diet high in β-cryptoxanthin may help ward off metabolic syndrome, liver dysfunction, insulin resistance, oxidative stress, low bone mass, and atherosclerosis.

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A Plant-Based Diet, Atherogenesis, and Coronary Artery Disease Prevention

Adherence to a plant-based dietary lifestyle may significantly cut down atherosclerosis and coronary artery disease (CAD) risk.

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Intensive lifestyle changes for reversal of coronary heart disease.

A lifestyle that involves the regular intake of low-fat vegetarian diets, aerobic exercise, and stress reduction may improve and reverse the conditions of coronary heart disease patients.

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Vascular endothelium and human disease.

Endothelial dysfunction may be a good indicator of the presence of various diseases, such as diabetes, stroke, cancer, and atherosclerosis.

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Intestinal microbiota metabolism and atherosclerosis.

Trimethylamine-N-oxide (TMAO), a metabolite produced by intestinal microbes, is associated with elevated atherosclerosis and cardiovascular disease risk.

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Intestinal microbiota metabolism of L-Carnitine, a nutrient in red meat, promotes atherosclerosis.

L-carnitine, a major component of red meat, may increase an individual’s susceptibility to cardiovascular disease, such as atherosclerosis, and adverse cardiac events, such as stroke and myocardial infarction.

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Compared with dietary monounsaturated and saturated fat, polyunsaturated fat protects African green monkey from coronary artery atherosclerosis.

High intake of polyunsaturated fat may lower the risk of developing coronary artery atherosclerosis in African green monkeys. This study evaluated the relationship between the consumption of monounsaturated, saturated, and polyunsaturated fat and coronary artery atherosclerosis development. Researchers fed 3 groups of African green monkeys with diets rich in saturated, monounsaturated, and polyunsaturated fats....
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Cholesterol crystals piercing the arterial plaque and intima trigger local and systemic inflammation.

The occurrence of local and systemic inflammation in atherosclerosis is linked to the plaque piercing action of cholesterol crystals.

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Cholesterol crystals cause mechanical damage to biological membranes: a proposed mechanism of plaque rupture and erosion leading to arterial thrombosis.

Cholesterol crystals may promote arterial thrombosis via arteriosclerotic plaque disruption and erosion.

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