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Soy isoflavone intake and breast cancer risk in Japan: from the Takayama study.

Increased consumption of soy and isoflavone may decrease breast cancer risk in postmenopausal Japanese women.

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Dietary patterns and breast cancer risk: a systematic review and meta-analysis.

Low breast cancer risk is associated with high intake of healthy foods.

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A meta-analysis of studies of dietary fats and breast cancer risk.

High consumption of fatty foods, such as meat, milk, and cheese, may increase breast cancer risk.

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Meat consumption among Black and White men and prostate cancer in the Cancer Prevention Study II Nutrition Cohort.

Increased intake of processed and unprocessed red meat is associated with high incidence of prostate cancer among Black men.

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Intestinal microbiota-dependent phosphatidylcholine metabolites, diastolic dysfunction, and adverse clinical outcomes in chronic systolic heart failure.

Increased dietary exposure to choline, betaine, and trimethylamine-N-oxide (TMAO) predisposes cardiovascular disease patients to adverse cardiac events, such heart failure.

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Impairment of endothelial function after a high-fat meal in patients with coronary artery disease.

Diets rich in fats may impair endothelial function in both coronary artery disease (CAD) patients and healthy individuals.

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Vascular endothelium and human disease.

Endothelial dysfunction may be a good indicator of the presence of various diseases, such as diabetes, stroke, cancer, and atherosclerosis.

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Postprandial angina pectoris: clinical and angiographic correlations.

Postprandial angina pectoris is associated with severe coronary artery disease.

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Longitudinal assessment of neurocognitive function after coronary-artery bypass surgery.

A decline in cognitive function is associated with coronary artery bypass surgery.

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Cholesterol crystals piercing the arterial plaque and intima trigger local and systemic inflammation.

The occurrence of local and systemic inflammation in atherosclerosis is linked to the plaque piercing action of cholesterol crystals.

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Cholesterol crystals cause mechanical damage to biological membranes: a proposed mechanism of plaque rupture and erosion leading to arterial thrombosis.

Cholesterol crystals may promote arterial thrombosis via arteriosclerotic plaque disruption and erosion.

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Cholesterol crystals and inflammation.

High serum concentrations of cholesterol crystals may elevate vascular inflammation in cardiovascular ailments and atherosclerosis risk.

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